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Adiponectin

Metabolic & Circadian Hormones
ADIPOQAdipoQAcrp30

Overview

Adiponectin, also known as ADIPOQ, AdipoQ, or Acrp30, is an endogenous hormone predominantly produced by adipose tissue. It belongs to the chemical class of proteins and is a significant component of the metabolic and circadian hormone category. Adiponectin circulates in the bloodstream and is involved in various metabolic processes. It is a 244-amino acid protein that forms multimers, which are crucial for its biological activity. Researchers have extensively studied adiponectin due to its role in metabolic regulation and its potential therapeutic implications. Adiponectin plays a critical role in regulating glucose levels and fatty acid breakdown. It is inversely correlated with body fat percentage in adults, and lower levels are associated with obesity, type 2 diabetes, and cardiovascular diseases. Researchers have found that adiponectin enhances insulin sensitivity and exhibits anti-inflammatory properties. It is also involved in energy homeostasis and has been a focus of research in metabolic syndrome and related disorders. The mechanism of action of adiponectin involves binding to its receptors, AdipoR1 and AdipoR2, which are widely expressed in various tissues. Activation of these receptors triggers AMP-activated protein kinase (AMPK) and peroxisome proliferator-activated receptor alpha (PPAR-α) pathways, leading to increased fatty acid oxidation and improved insulin sensitivity. Researchers have observed that these pathways are crucial for the hormone's metabolic effects. The pharmacokinetic properties of adiponectin are not fully characterized, but it is known to have a relatively short circulating half-life, estimated to be around 70 minutes. Its metabolism and bioavailability by different routes are not well-documented, as it is primarily studied in its endogenous form. Clinical use of adiponectin as a therapeutic agent is still under investigation, and it is not currently approved for any specific medical treatment. Researchers are exploring its potential in treating metabolic disorders, but it remains in the research phase without regulatory approval for clinical use.

Mechanism of Action

Adiponectin acts on AdipoR1 and AdipoR2 receptors, activating AMPK and PPAR-α pathways. This leads to increased fatty acid oxidation and enhanced insulin sensitivity, contributing to its metabolic effects.

Half-Life & Pharmacokinetics

EndogenousCirculating half-life ~70 minutes

Pharmacokinetic properties are primarily studied in the context of its endogenous form.

Storage

Temperature

Refrigerate at 2-8°C

Light

Protect from light

Form

Aqueous solution: use within specified time after preparation

Notes

Ensure stability by storing under recommended conditions.

Solubility

Adiponectin is soluble in aqueous solutions, relevant for its biological activity studies.

Legal Status

🇩🇪DE

Unknown

🇺🇸US

Unknown

🇦🇺AU

Unknown

🇬🇧UK

Unknown

Legal status information is provided for general reference only and may not reflect the most current regulatory changes. Always verify with official government sources before making any decisions.

10 Research Publications

Macrophage-derived amphiregulin induces myofibroblast transition in adipogenic lineage precursors near Staphylococcus aureus abscess in bone marrow.

Unknown

Nature communications · 2025

Researchers found that a substance produced by immune cells, called amphiregulin, plays a role in transforming certain cells in the bone marrow into myofibroblasts near infections caused by Staphylococcus aureus. This process may help explain why these bacterial infections can persist in the body.

  • Amphiregulin is produced by macrophages in response to Staphylococcus aureus abscesses.
  • This substance induces a change in adipogenic lineage precursors to myofibroblasts.
  • Understanding this mechanism could provide insights into persistent skeletal infections.
PubMed

The Role of Adiponectin and

Review

Genes · 2025

Researchers observed that metabolic syndrome, which increases the risk of diabetes and heart disease, is influenced by adiponectin, a substance produced by fat tissue. This protein helps manage blood sugar and fat levels and may protect blood vessels from damage.

  • Metabolic syndrome is a cluster of conditions that raises the risk of diabetes and heart disease.
  • Adiponectin plays a key role in regulating glucose and fat levels in the body.
  • Adiponectin may help protect blood vessels from harmful changes.
PubMed

Schnurri-3 controls osteogenic fate of Adipoq-lineage progenitors in bone marrow.

Unknown

Journal of orthopaedic translation · 2024

Researchers observed that a specific gene, Schnurri-3, plays a significant role in controlling the ability of certain bone marrow cells to help maintain and repair bone. By targeting this gene, they found a potential new approach for treating conditions related to bone loss, such as osteoporosis and fractures.

  • Schnurri-3 regulates the fate of Adipoq-lineage progenitors in bone marrow.
  • Targeting Schnurri-3 could enhance bone repair and maintenance.
  • This research highlights new therapeutic strategies for bone loss-related disorders.
PubMed

A Meta-analysis of ADIPOQ rs2241766 polymorphism association with type 2 diabetes.

Meta-Analysis

Journal of diabetes and metabolic disorders · 2022

Researchers found a link between a specific genetic variation in the adiponectin gene (ADIPOQ rs2241766) and an increased risk of developing type 2 diabetes. While some studies showed a strong association, others did not find significant results, indicating more research is needed to fully understand this relationship.

  • ADIPOQ rs2241766 polymorphism is associated with an increased risk of type 2 diabetes.
  • Some studies reported a significant increase in risk, while others showed no significant association.
  • Overall, the genetic variation may contribute to the likelihood of developing type 2 diabetes.
PubMed

Evaluation of the frequency of ADIPOQ c.45 T>G and ADIPOQ c.276 G>T polymorphisms in adiponectin coding gene in girls with anorexia nervosa.

Meta-Analysis

Endokrynologia Polska · 2021

Researchers found that genetic variations in the ADIPOQ gene do not appear to influence the likelihood of developing anorexia nervosa in girls. However, they observed that girls with anorexia who have certain genetic types may have higher levels of adiponectin, which could indicate better insulin sensitivity and adaptation to malnutrition.

  • Genetic variations in ADIPOQ do not affect the risk of anorexia nervosa.
  • Girls with anorexia and specific ADIPOQ genotypes have higher adiponectin levels.
  • Higher adiponectin levels may suggest better insulin sensitivity in girls with anorexia.
PubMed

Adiponectin GWAS loci harboring extensive allelic heterogeneity exhibit distinct molecular consequences.

Unknown

PLoS genetics · 2020

Researchers found that specific genetic regions associated with adiponectin, a hormone important for regulating blood sugar, contain multiple signals that influence its levels. They identified distinct genetic variations that affect the expression of different genes related to adiponectin, revealing complex mechanisms behind these associations.

  • Four genetic regions were linked to adiponectin levels in a study of 9,262 men.
  • Two regions showed multiple genetic signals affecting different genes.
  • Distinct genetic variations influenced the production of adiponectin through various molecular mechanisms.
PubMed

Associations between ADIPOQ polymorphisms and coronary artery disease: a meta-analysis.

Meta-Analysis

BMC cardiovascular disorders · 2019

Researchers found that specific genetic variations in the adiponectin gene (ADIPOQ) are linked to an increased risk of coronary artery disease (CAD). They observed that one variation, rs2241766, is associated with CAD in multiple populations, while another variation, rs1501299, is particularly linked to CAD in East Asians.

  • The rs2241766 variation is associated with CAD risk in Caucasians, East Asians, and South Asians.
  • The rs1501299 variation is significantly associated with CAD risk in East Asians.
  • These findings suggest that certain genetic factors influence CAD susceptibility in different ethnic groups.
PubMed

Association of ADIPOQ and ADIPOR variants with risk of colorectal cancer: A meta-analysis.

Meta-Analysis

Journal of Huazhong University of Science and Technology. Medical sciences = Hua zhong ke ji da xue xue bao. Yi xue Ying De wen ban = Huazhong keji daxue xuebao. Yixue Yingdewen ban · 2017

Researchers found that specific genetic variations in the ADIPOQ and ADIPOR genes are associated with an increased risk of colorectal cancer. The study analyzed data from over 6,000 cancer patients and highlighted that certain gene variants may interact with factors related to insulin resistance, influencing cancer risk in different populations.

  • ADIPOQ variants rs2241766, rs1501299, and ADIPOR variant rs1342387 are linked to colorectal cancer risk.
  • Different populations showed varying associations with specific gene variants.
  • Insulin resistance may enhance the risk of colorectal cancer in individuals with certain genetic variants.
PubMed

Association of adiponectin gene (ADIPOQ) rs2241766 polymorphism with obesity in adults: a meta-analysis.

Meta-Analysis

PloS one · 2014

Researchers found that a specific genetic variation in the adiponectin gene (ADIPOQ-rs2241766) may increase the risk of obesity in adults, particularly among Chinese populations. However, this association was not observed in non-Chinese groups, indicating that genetic factors influencing obesity may vary by region.

  • The GG genotype of ADIPOQ-rs2241766 is linked to a higher obesity risk.
  • The GT genotype shows a borderline increased risk for obesity.
  • The association is significant in Chinese studies but not in non-Chinese studies.
PubMed

Adiponectin (ADIPOQ) rs2241766 G/T polymorphism is associated with risk of cancer: evidence from a meta-analysis.

Meta-Analysis

Tumour biology : the journal of the International Society for Oncodevelopmental Biology and Medicine · 2013

Researchers found that a specific genetic variation in the adiponectin gene (rs2241766 G/T) may be linked to a lower risk of developing cancer, particularly in certain populations. However, another variation (rs266729 G/C) did not show any significant association with cancer risk.

  • The rs2241766 G/T variation was associated with a decreased cancer risk in population-based studies.
  • The rs266729 G/C variation showed no significant link to cancer risk.
  • The analysis included over 23,000 participants across multiple studies.
PubMed

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This page is for informational and research purposes only. All information is based on published scientific literature. Nothing on this page constitutes medical advice or replaces consultation with a qualified healthcare professional.