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Prolactin

Pituitary & Trophic Hormones
PRLLuteotropinLuteotropic Hormone

Overview

Prolactin (PRL), also known as luteotropin or luteotropic hormone, is an endogenous peptide hormone produced primarily by the anterior pituitary gland. It belongs to the protein hormone class and is encoded by the PRL gene in humans. Prolactin is synthesized and secreted by lactotroph cells in the pituitary and plays a crucial role in various physiological processes. Researchers have extensively studied prolactin for its involvement in lactation, reproductive functions, and its broader role in homeostasis. The primary physiological roles of prolactin include the stimulation of milk production in mammals, regulation of the immune system, and modulation of reproductive behaviors. Research areas have expanded to include its effects on metabolism, osmoregulation, and its potential role in certain pathologies such as prolactinomas and hyperprolactinemia. Prolactin exerts its effects through binding to the prolactin receptor, a member of the cytokine receptor superfamily. This interaction activates the JAK2-STAT signaling pathway, leading to the transcription of target genes involved in milk production and other physiological processes. Researchers have found that prolactin's mechanism of action is complex, involving cross-talk with other hormonal pathways. The pharmacokinetic properties of prolactin indicate a circulating half-life of approximately 15-20 minutes. It is primarily metabolized in the liver and kidneys, with its bioavailability being largely dependent on endogenous secretion rather than exogenous administration. Clinical use of prolactin is limited, as it is not typically administered as a therapeutic agent. However, its levels are measured in clinical settings to diagnose and manage conditions like prolactinomas and infertility. Regulatory standing for prolactin as a therapeutic agent is not applicable, as it is not used in pharmacological treatments.

Mechanism of Action

Prolactin acts on the prolactin receptor, which is part of the cytokine receptor superfamily. Upon binding, it activates the JAK2-STAT signaling pathway, leading to gene transcription that supports lactation and other physiological functions.

Molecular Data

FormulaC160H194N22O54S4
Molecular Weight3417.6 g/mol
CAS Number9002-62-4
PubChem CID168266256

Half-Life & Pharmacokinetics

EndogenousCirculating half-life ~15-20 minutes

Prolactin is primarily studied in its endogenous form, with pharmacokinetics focused on natural secretion and metabolism.

Storage

Temperature

Refrigerate at 2-8°C

Light

Protect from light

Form

Aqueous solution: use within specified period after opening

Notes

Storage conditions are critical for maintaining the stability of prolactin assays used in clinical diagnostics.

Solubility

Prolactin is soluble in water, which is relevant for its formulation in diagnostic assays.

Legal Status

🇩🇪DE

Data limited

🇺🇸US

Prolactin is not a controlled substance and does not require FDA approval as it is not used as a therapeutic agent.

🇦🇺AU

Data limited

🇬🇧UK

Data limited

Legal status information is provided for general reference only and may not reflect the most current regulatory changes. Always verify with official government sources before making any decisions.

10 Research Publications

Prolactin: structure, receptors, and functions.

Review

Reviews in endocrine & metabolic disorders · 2024

Researchers observed that prolactin (PRL), a protein produced mainly in the pituitary gland, plays a crucial role in reproduction and nurturing behaviors. Additionally, they found that PRL is involved in various health issues, including migraines, metabolic regulation, and certain diseases like cancer, highlighting its complex functions in the body.

  • PRL regulates reproductive functions and maternal behaviors.
  • PRL is linked to migraines and metabolic health.
  • PRL may influence inflammatory diseases and cancer.
PubMed

The role of prolactin/vasoinhibins in cardiovascular diseases.

Review

Animal models and experimental medicine · 2023

Researchers observed that prolactin, a hormone produced in the pituitary gland, has two main forms that influence blood vessel formation in different ways. They found that these forms can either promote or inhibit vascular health, which is important for understanding their roles in heart and blood vessel diseases.

  • Prolactin has two main forms: full-length prolactin and vasoinhibins.
  • These forms can stimulate or inhibit blood vessel formation.
  • Their effects on heart health can be opposite, highlighting their complex roles in cardiovascular diseases.
PubMed

The Involvement of Prolactin in Stress-Related Disorders.

Review

International journal of environmental research and public health · 2023

Researchers observed that prolactin (PRL) plays a significant role in managing stress responses, especially during pregnancy and breastfeeding. While elevated PRL levels are beneficial in these situations, they can lead to serious health issues when occurring outside of pregnancy, such as affecting ovulation. The study highlights the complex effects of PRL on brain function and emotional well-being, particularly in relation to stress-related disorders.

  • PRL helps regulate stress responses during pregnancy and lactation.
  • Elevated PRL levels can lead to health issues like ovulation suppression.
  • The hormone's effects on the brain are important for maternal emotional well-being.
PubMed

The beneficial metabolic actions of prolactin.

Review

Frontiers in endocrinology · 2022

Researchers observed that prolactin (PRL) plays a beneficial role in maintaining metabolic health. Moderate levels of PRL are linked to improved insulin sensitivity and reduced risks of conditions like fatty liver and type 2 diabetes in both animals and humans. The study suggests that increasing PRL levels could be a potential strategy for treating metabolic diseases.

  • Moderate PRL levels are beneficial for metabolism.
  • High and low PRL levels can lead to negative health outcomes.
  • PRL positively affects key metabolic organs like the pancreas and liver.
PubMed

Prolactin actions.

Review

Journal of molecular endocrinology · 2014

Researchers observed that recent advancements in molecular genetics have improved our understanding of prolactin (PRL) and its effects on both cells and the body as a whole. The review highlights significant progress in understanding how PRL functions in humans and discusses how these findings may shape future research directions.

  • Advancements in molecular genetics enhance understanding of PRL actions.
  • New insights focus on human-specific effects of PRL.
  • Future research priorities are influenced by these discoveries.
PubMed

[Prolactin and serotonin].

Review

Vestnik Rossiiskoi akademii meditsinskikh nauk · 2014

Researchers found that prolactin (PRL) plays a role in breast and prostate cancer risk and that certain genetic variations in its receptor may be linked to breast tumors. They also observed that PRL's effects can be inhibited by synthesized analogues and that the nervous system influences its secretion. The study highlights potential clinical applications of PRL in breast cancer therapy.

  • Prolactin is a risk factor for breast and prostate cancer.
  • Genetic variations in the prolactin receptor are associated with breast tumors.
  • Synthesized analogues of prolactin can inhibit its effects.
PubMed

S179D prolactin: antagonistic agony!

Review

Molecular and cellular endocrinology · 2007

Researchers observed that a modified form of prolactin, known as S179D prolactin, may offer new insights into how prolactin interacts with its receptors. They propose that instead of traditional antagonists, therapies that signal in a more nuanced way could be more effective. This approach introduces the concept of selective prolactin receptor modulators (SPeRMs).

  • S179D prolactin provides insights into prolactin-receptor interactions.
  • Therapies that signal antagonistically may be more effective than pure antagonists.
  • The term selective prolactin receptor modulator (SPeRM) is proposed for certain therapeutic agents.
PubMed

Reproductive role of prolactin.

Review

Reproduction (Cambridge, England) · 2007

Researchers observed that prolactin (PRL), a hormone primarily known for its role in milk production, is also crucial for reproduction. They found that disruptions in PRL and its receptor can lead to reproductive issues. The review highlights advancements in understanding PRL's functions and its implications in reproductive health over the past two decades.

  • Prolactin is essential for lactation and reproduction.
  • Deficiencies in prolactin or its receptor lead to reproductive failures.
  • Recent studies have improved our understanding of prolactin's biological actions.
PubMed

Prolactin receptor antagonists.

Review

Endocrine · 1998

Researchers observed that prolactin is produced not only by the pituitary gland but also by various tissues in the body. They found that prolactin receptor antagonists could help understand the local effects of prolactin and may offer new treatment options for diseases related to its production. The study reviews current knowledge on prolactin signaling and the development of antagonists to further explore these mechanisms.

  • Prolactin is produced by both the pituitary gland and other tissues.
  • Prolactin receptor antagonists can help analyze local effects of prolactin.
  • These antagonists may lead to new treatments for diseases linked to prolactin.
PubMed

Prolactin variants.

Unknown

Trends in endocrinology and metabolism: TEM · 1992

Researchers found that prolactin (PRL), a hormone with many functions in the body, exists in different forms that can have varying effects. This diversity in PRL's structure may help explain why it can trigger such a wide range of biological responses.

  • Prolactin has multiple structural forms.
  • Different forms of prolactin have varying effects.
  • The diversity in prolactin may contribute to its various biological actions.
PubMed

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This page is for informational and research purposes only. All information is based on published scientific literature. Nothing on this page constitutes medical advice or replaces consultation with a qualified healthcare professional.