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SS-31

Anti-Aging & Longevity
ElamipretideMTP-131Bendavia

Overview

SS-31, also known as Elamipretide, MTP-131, or Bendavia, is a synthetic tetrapeptide compound developed for its potential therapeutic effects on mitochondrial function. It is composed of the amino acids D-arginine, dimethyltyrosine, lysine, and phenylalanine, forming a small, water-soluble peptide. The compound was designed to target and penetrate mitochondria, with synthesis methods focusing on enhancing its stability and bioavailability. Researchers have primarily investigated SS-31 in the context of mitochondrial dysfunction, which is implicated in a variety of age-related diseases and conditions. Studies have shown that SS-31 can improve mitochondrial function, reduce oxidative stress, and enhance cellular energy production, leading to potential benefits in conditions such as heart failure, kidney disease, and neurodegenerative disorders. The mechanism of action of SS-31 involves its interaction with cardiolipin, a phospholipid found in the inner mitochondrial membrane. By binding to cardiolipin, SS-31 stabilizes mitochondrial membranes, reduces the production of reactive oxygen species, and enhances ATP synthesis. This action helps to preserve mitochondrial function and protect cells from damage. Pharmacokinetic studies of SS-31 indicate that it has a relatively short half-life, with rapid distribution and clearance from the body. The compound is stable in its lyophilized form and shows good solubility in aqueous solutions, although its oral bioavailability is poor. Current research on SS-31 is focused on its therapeutic potential in clinical trials for various conditions, but it has not yet received regulatory approval as a medicinal product. Its status remains investigational, with ongoing studies to further elucidate its efficacy and safety profile.

Mechanism of Action

SS-31 exerts its effects by selectively binding to cardiolipin in the inner mitochondrial membrane. This interaction stabilizes the mitochondrial structure, reduces oxidative stress, and enhances ATP production, thereby improving overall mitochondrial function.

Molecular Data

FormulaC32H49N9O5
Molecular Weight639.8 g/mol
CAS Number736992-21-5
PubChem CID11764719

Half-Life

Subcutaneous~4 hours
Intravenous~30 minutes
IntranasalNot applicable
OralPoor bioavailability

SS-31 is rapidly distributed and cleared, with a short half-life that varies depending on the route of administration.

Storage

Lyophilized

Stable for 2+ years at -20°C, 12 months at 4°C

Reconstituted

Use within 14 days when refrigerated at 4°C

Avoid

Avoid repeated freeze-thaw cycles, direct light

Solvent

Bacteriostatic water or sterile saline recommended

Solubility

SS-31 is highly soluble in water and aqueous solutions.

Legal Status

🇩🇪DE

Not approved as a medicinal product. Not a controlled substance. Sale as research chemical is a legal grey area.

🇺🇸US

Not approved by the FDA as a medicinal product. Not scheduled by the DEA.

🇦🇺AU

Not approved by the TGA as a medicinal product.

🇬🇧UK

Not approved by the MHRA as a medicinal product.

Legal status information is provided for general reference only and may not reflect the most current regulatory changes. Always verify with official government sources before making any decisions.

10 Research Publications

Elamipretide: First Approval.

Review

Drugs · 2026

Researchers observed that elamipretide, a new treatment developed for mitochondrial disorders, received accelerated approval in the USA for improving muscle strength in patients with Barth syndrome. This marks a significant milestone as it is the first specific treatment approved for this rare genetic condition. Additionally, elamipretide is being studied for other conditions related to mitochondrial dysfunction.

  • Elamipretide became the first approved treatment for Barth syndrome, a rare genetic disorder.
  • The approval was granted in September 2025 for both adult and pediatric patients weighing 30 kg or more.
  • Elamipretide is also under investigation for other conditions, including dry age-related macular degeneration and mitochondrial myopathies.
PubMed

Contemporary insights into elamipretide's mitochondrial mechanism of action and therapeutic effects.

Review

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie · 2025

Researchers reviewed the effects of elamipretide, a peptide that targets mitochondria, which are crucial for energy production in cells. They found that elamipretide improves mitochondrial function and structure, showing promise in treating various mitochondrial disorders based on recent studies and clinical trials.

  • Researchers observed that elamipretide interacts with cardiolipin, a key component of mitochondrial membranes, enhancing mitochondrial performance.
  • The review highlighted elamipretide's potential benefits in animal models of heart and muscle diseases, as well as eye conditions.
  • Recent clinical trials have provided insights into elamipretide's effects on conditions like Barth syndrome and age-related macular degeneration.
PubMed

Mitochondrial Dysfunction: The Silent Catalyst of Kidney Disease Progression.

Review

Cells · 2025

Researchers observed that mitochondrial dysfunction plays a crucial role in the development of kidney diseases, including acute kidney injury and chronic kidney disease. The study highlights how disruptions in mitochondrial function can lead to increased inflammation and oxidative stress, worsening kidney health.

  • Mitochondrial dysfunction is a key factor in the progression of acute kidney injury and chronic kidney disease.
  • Excessive fission of mitochondria contributes to cell death and inflammation in kidney injury models.
  • In chronic kidney disease, ongoing mitochondrial issues lead to oxidative stress and changes in kidney cell metabolism.
PubMed

SS-31@Fer-1 Alleviates ferroptosis in hypoxia/reoxygenation cardiomyocytes via mitochondrial targeting.

Unknown

Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie · 2025

Researchers found that a new compound, SS-31@Fer-1, can protect heart cells from damage caused by a lack of oxygen followed by reoxygenation, a condition often seen in heart attacks. This study observed that SS-31@Fer-1 improves cell survival and maintains iron balance in heart cells, which is crucial for their health.

  • SS-31@Fer-1 significantly improved the survival of heart cells under stress from hypoxia/reoxygenation.
  • The compound helped maintain iron balance and reduced harmful oxidative stress in the cells.
  • Researchers noted enhanced mitochondrial function and integrity in heart cells treated with SS-31@Fer-1.
PubMed

Elamipretide: A Review of Its Structure, Mechanism of Action, and Therapeutic Potential.

Review

International journal of molecular sciences · 2025

Researchers reviewed the potential of Elamipretide, a compound that targets mitochondria, which are crucial for energy production in cells. They observed that Elamipretide can stabilize mitochondrial structures, reduce oxidative stress, and enhance energy production, showing promise in treating various health conditions linked to mitochondrial dysfunction.

  • Elamipretide selectively binds to a lipid in mitochondria, stabilizing their structure.
  • Preclinical studies indicated its protective effects in heart failure and neurodegenerative diseases.
  • Clinical trials are underway to further explore its therapeutic potential and safety.
PubMed

Efficacy and Safety of Elamipretide in Individuals With Primary Mitochondrial Myopathy: The MMPOWER-3 Randomized Clinical Trial.

Human

Neurology · 2023

Researchers studied the effects of elamipretide on individuals with primary mitochondrial myopathy, a genetic disorder affecting muscle function and energy production. The study found that elamipretide did not improve walking distance or fatigue levels compared to a placebo over 24 weeks, although it was well-tolerated by participants.

  • The study did not meet its primary goals of improving walking distance or fatigue in participants taking elamipretide.
  • Participants receiving elamipretide showed no significant difference in the 6-minute walk test compared to those receiving a placebo.
  • Elamipretide was well-tolerated, indicating it may be safe for use in this patient population.
PubMed

A ROS-Responsive Liposomal Composite Hydrogel Integrating Improved Mitochondrial Function and Pro-Angiogenesis for Efficient Treatment of Myocardial Infarction.

Unknown

Advanced healthcare materials · 2022

Researchers developed a special hydrogel that delivers drugs to heart cells affected by a heart attack. This study observed that the hydrogel improves heart cell function by targeting damaged mitochondria and promoting the growth of new blood vessels. The findings suggest a promising approach for enhancing recovery after a heart attack.

  • Researchers found that the hydrogel releases drugs that reduce harmful reactive oxygen species in heart cells.
  • The study observed that the hydrogel improves mitochondrial function in heart cells.
  • Researchers noted that the hydrogel promotes angiogenesis, which is the formation of new blood vessels.
PubMed

Targeting mitochondrial dysfunction with elamipretide.

Review

Heart failure reviews · 2022

Researchers reviewed the potential of elamipretide, a new treatment targeting mitochondria, to improve heart function in patients with heart failure. They found that elamipretide may enhance heart performance and prevent heart damage in animal models, with early clinical trials showing promising results and no severe side effects.

  • Researchers observed that elamipretide increased heart function in dog models with heart failure.
  • The treatment prevented heart structure changes in rat models.
  • Early clinical trials indicated elamipretide was safe and showed improvements in heart blood flow.
PubMed

Potential Therapeutic Candidates for Age-Related Macular Degeneration (AMD).

Review

Cells · 2021

Researchers reviewed potential new treatments for Age-related Macular Degeneration (AMD), a major cause of blindness linked to aging. They identified several promising therapeutic candidates that may protect retinal cells and combat the disease's progression.

  • Researchers observed that retinal aging involves various changes, including inflammation and mitochondrial dysfunction, contributing to AMD.
  • The study highlighted several potential therapeutic candidates, including APE/REF-1 and Celecoxib, that may offer cytoprotection in AMD.
  • Limited treatment options for AMD underscore the importance of exploring new therapeutic targets to improve patient outcomes.
PubMed

Elamipretide (SS-31) improves mitochondrial dysfunction, synaptic and memory impairment induced by lipopolysaccharide in mice.

Animal

Journal of neuroinflammation · 2019

Researchers studied the effects of elamipretide, a mitochondrion-targeted antioxidant, on memory impairment caused by inflammation in mice. They found that elamipretide improved learning and memory performance by reducing mitochondrial dysfunction and oxidative stress, suggesting its potential for addressing cognitive issues related to neuroinflammation.

  • Elamipretide significantly improved memory performance in mice affected by lipopolysaccharide-induced inflammation.
  • The treatment reduced oxidative stress and mitochondrial dysfunction in the brain.
  • Elamipretide enhanced the signaling of brain-derived neurotrophic factor, which is important for synaptic health.
PubMed

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This page is for informational and research purposes only. All information is based on published scientific literature. Nothing on this page constitutes medical advice or replaces consultation with a qualified healthcare professional.